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nmda receptor antagonist mk801  (Biotrend Chemicals)

 
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    Biotrend Chemicals nmda receptor antagonist mk801
    Nmda Receptor Antagonist Mk801, supplied by Biotrend Chemicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/nmda+receptor+antagonist+mk801/mk+801/pm29438741-74-1-8
    Average 90 stars, based on 1 article reviews
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    other:

    Article Title: Exposure to a novel context following contextual fear conditioning enhances the induction of hippocampal long-term potentiation.
    Article Snippet: The prior behavioral experience of an animal can influence the direction and the probability of long-term plasticity induced at the activated synapses.. In the present study, we compared alterations in long-term potentiation in the rat CA1 of the hippocampus following post-fear conditioning exposure to the conditioning context vs. a novel context.. Furthermore, we examined whether the alterations in long-term potentiation are dependent on the prior formation of context–shock fear memory association.

    Article Title: Different mechanisms underlie stress-induced changes in plasticity and metaplasticity in the prefrontal cortex of juvenile and adult animals: Emotional-induced metaplasticity in the prefrontal cortex.
    Article Snippet: Metaplasticity is the dynamic regulation of the ability to induce activity-dependent synaptic plasticity and is governed by the prior history of the synapses.. Previous reports by others and us have shown that behavioral stress induces a form of emotional metaplasticity that affects the ability to induce LTP in the subiculum–medial prefrontal cortex pathway, which depends on NMDA receptors (NMDAr).. However, studies addressing the effects of stress on LTP and metaplasticity have mainly focused on the adult animal.



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    NMDA receptor-dependent FVP (A) VEPs in NR2A knockout mice on days 1 and 6 (contralateral eye stimulation). (B) Absence of changes in VEP peaks (day 1 vs. day 6) in NR2A knockout mice after visual stimulation ( N = 6 mice, 12 recording sites, 24 recordings of contralateral or ipsilateral eye stimulation). Mann–Whitney test, N1: p = 0.5775; P1: p = 0.5844; N2: p = 0.4964. (C) VEPs of wild-type mice injected with <t>MK801</t> intraperitoneally (0.5 mg/kg body weight) (day 1 vs. day 6, contralateral eye stimulation) 30 min before the start of visual stimulation. (D) Absence of FVP (day 1 vs. day 6) in MK801 treated wild type mice after visual stimulation. VEP peak N2 amplitudes ( N = 6 mice, 12 recording sites, 24 recordings of contralateral or ipsilateral eye stimulation). Mann–Whitney test, N1: p = 0.1305; P1: p = 0.4552, N2: p = 0.1060. (E) Impaired rapid potentiation in NR2A KO mice. Visual response (area under waveform) during the initial stimulation (0–10 pulses) vs. late stimulation (190–200 pulses) on day 1. Paired t -test, p = 0.924, 6 mice, t = 0.1002, df = 5.
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    NMDA receptor-dependent FVP (A) VEPs in NR2A knockout mice on days 1 and 6 (contralateral eye stimulation). (B) Absence of changes in VEP peaks (day 1 vs. day 6) in NR2A knockout mice after visual stimulation ( N = 6 mice, 12 recording sites, 24 recordings of contralateral or ipsilateral eye stimulation). Mann–Whitney test, N1: p = 0.5775; P1: p = 0.5844; N2: p = 0.4964. (C) VEPs of wild-type mice injected with MK801 intraperitoneally (0.5 mg/kg body weight) (day 1 vs. day 6, contralateral eye stimulation) 30 min before the start of visual stimulation. (D) Absence of FVP (day 1 vs. day 6) in MK801 treated wild type mice after visual stimulation. VEP peak N2 amplitudes ( N = 6 mice, 12 recording sites, 24 recordings of contralateral or ipsilateral eye stimulation). Mann–Whitney test, N1: p = 0.1305; P1: p = 0.4552, N2: p = 0.1060. (E) Impaired rapid potentiation in NR2A KO mice. Visual response (area under waveform) during the initial stimulation (0–10 pulses) vs. late stimulation (190–200 pulses) on day 1. Paired t -test, p = 0.924, 6 mice, t = 0.1002, df = 5.

    Journal: Frontiers in Neural Circuits

    Article Title: Rapid and cumulative adult plasticity in the mouse visual cortex

    doi: 10.3389/fncir.2025.1537305

    Figure Lengend Snippet: NMDA receptor-dependent FVP (A) VEPs in NR2A knockout mice on days 1 and 6 (contralateral eye stimulation). (B) Absence of changes in VEP peaks (day 1 vs. day 6) in NR2A knockout mice after visual stimulation ( N = 6 mice, 12 recording sites, 24 recordings of contralateral or ipsilateral eye stimulation). Mann–Whitney test, N1: p = 0.5775; P1: p = 0.5844; N2: p = 0.4964. (C) VEPs of wild-type mice injected with MK801 intraperitoneally (0.5 mg/kg body weight) (day 1 vs. day 6, contralateral eye stimulation) 30 min before the start of visual stimulation. (D) Absence of FVP (day 1 vs. day 6) in MK801 treated wild type mice after visual stimulation. VEP peak N2 amplitudes ( N = 6 mice, 12 recording sites, 24 recordings of contralateral or ipsilateral eye stimulation). Mann–Whitney test, N1: p = 0.1305; P1: p = 0.4552, N2: p = 0.1060. (E) Impaired rapid potentiation in NR2A KO mice. Visual response (area under waveform) during the initial stimulation (0–10 pulses) vs. late stimulation (190–200 pulses) on day 1. Paired t -test, p = 0.924, 6 mice, t = 0.1002, df = 5.

    Article Snippet: The NMDA receptor antagonist MK801 (FUJIFILM Wako Pure Chemical Corporation, Osaka, Japan) was dissolved in saline (0.1 mg/mL) and administered intraperitoneally (0.5 mg/kg) 15 min before daily recording sessions for 6 consecutive days.

    Techniques: Knock-Out, MANN-WHITNEY, Injection